| REVIEW ARTICLE: REPUBLICATION |
|
| Year : 2018 | Volume
: 8
| Issue : 2 | Page : 73-77 |
|
|
Portal vein thrombosis: What surgeons need to know
Ricardo Quarrie1, Stanislaw P Stawicki2
1 Department of Surgery, The Ohio State University Medical Center, Columbus, OH, USA
2 Department of Surgery, The Ohio State University Medical Center, Columbus, OH; OPUS 12 Foundation, Bethlehem, PA, USA
Correspondence Address:
Dr. Stanislaw P Stawicki
St. Luke's University Health Network, Bethlehem, Pennsylvania, 18015
USA
 Source of Support: None, Conflict of Interest: None  | Check |
DOI: 10.4103/IJCIIS.IJCIIS_71_17
|
|
|
Key points: (a) The lifetime risk of portal vein thrombosis (PVT) is approximately 1%; (b) The portal vein is formed by the union of the splenic and superior mesenteric veins posterior to the pancreas; (c) Imaging modalities most frequently used to diagnose PVT include sonography, computed tomography, and magnetic resonance imaging; (d) Malignancy, hepatic cirrhosis, surgical trauma, and hypercoagulable conditions are the most common risk factors for the development of PVT; (e) PVT eventually leads to the formation of numerous collateral vessels around the thrombosed portal vein; (f) First-line treatment for PVT is therapeutic anticoagulation—it helps prevent the progression of the thrombotic process; (g) Other therapeutic options include surgery and interventional radiographic procedures including mechanical thrombectomy and thrombolysis; (h) Portal biliopathy is a clinicopathologic entity characterized by biliary abnormalities due to portal hypertension secondary to PVT and appears to be more common in cases of extrahepatic PVT.
Republished with permission from: Quarrie R, Stawicki SP. Portal vein thrombosis: What surgeons need to know. OPUS 12 Scientist 2008;2(3):30-33.
|
|
|
|
| [FULL TEXT] [PDF]* |
|
 |
|
